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Adult mice with no previous exposure, low exposure, and high exposure to acetaminophen were given a dose 30 min prior to being placed on a hot plate

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Research in the roles of GHK-Cu suggests its actions may be mediated partially via an expression of transforming growth factor Beta

These effects reinforce the idea of GHK-Cu as a global repair signal

However, the therapeutic efficacy of these inhibitors in fatty liver disease remains to be established, underscoring the necessity for further investigation